451 Modeling darier disease using gene-edited human keratinocytes and organotypic epidermis to identify therapeutic targets

نویسندگان

چکیده

Darier disease(DD) is a genetic disorder characterized by impaired intercellular adhesion and aberrant differentiation of keratinocytes, leading to hyperkeratotic papules epidermal erosions. While mutations underlying DD are known target SERCA2, an endoplasmic reticulum(ER) calcium pump, current therapies for this chronic, painful skin limited some, such as oral retinoids, may pose significant long-term risks. Unfortunately, mice lacking SERCA2 do not replicate the cutaneous findings seen in human patients. Thus, we aimed establish vitro model improve our understanding its pathogenesis downstream deficiency reveal new potential therapeutic strategies. TERT-immortalized keratinocytes were subjected CRISPR/Cas9 gene editing generate cells deficient SERCA2. Transduction with GCaMP, cytosolic sensor, allowed us demonstrate augmented spikes intracellular SERCA2-depleted upon exposure elevated extracellular calcium. Culturing SERCA2-deficient organotypic epidermis revealed features histopathology including dyskeratosis reduced adhesion. To elucidate pathogenic drivers resulting from deficiency, performed RNA sequencing heterozygous or homozygous loss dysregulated homeostasis due depletion expected disrupt numerous cellular pathways, preliminary analysis expression profiles alterations potentially druggable targets, regulators cornification, adhesion, homeostasis, inflammatory responses, cell death. Further studies ongoing assess whether dampening effects using existing pharmacologic compounds can effectively reverse pathology epidermis.

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ژورنال

عنوان ژورنال: Journal of Investigative Dermatology

سال: 2022

ISSN: ['1523-1747', '0022-202X']

DOI: https://doi.org/10.1016/j.jid.2022.05.460